Catalysts function by providing an alternate reaction mechanism that has a lower activation energy than would be found in the absence of the catalyst. In some cases, the catalyzed mechanism may include additional steps.In a article, 19977-51-6, molcular formula is C8H12BrN, introducing its new discovery. Formula: C8H12BrN
Reaction of [Ru3(CO)9{mu3-eta1,kappa1,kappa2-PhP(C6H4)CH2PPh}] (1) with tri(2-thienyl)phosphine (PTh3) in refluxing THF afforded [Ru3(CO)9(PTh3)(mu-dpbm)] (3) {dpbm = PhP(C6H4)(CH2)PPh} and [Ru3(CO)6(mu-CO)2{mu-kappa1,eta1-PTh2(C4H2S)}{mu3-kappa1,kappa2-Ph2PCH2PPh}] (5) in 18% and 12% yields, respectively, while a similar reaction with tri(2-furyl)phosphine (PFu3) gave [Ru3(CO)9(PFu3)(mu-dpbm)] (4) and [Ru3(CO)7(mu-eta1,eta2-C4H3O)(mu-PFu2){mu3-eta1,kappa1,kappa2-PhP(C6H4)CH2PPh}] (6) in 24% and 27% yields, respectively. Compounds 2 and 4 are phosphine adducts of 1 in which the diphosphine ligand is transformed into 1,3-diphenyl-2,3-dihydro-1H-1,3-benzodiphosphine (dpbm) via phosphorus-carbon bond formation. Cluster 5 results from metalation of a thienyl ring, the cleaved proton being transferred to the diphosphine. Carbon-phosphorus bond cleavage of a PFu3 ligand is observed in 6 to afford a phosphido-bridge and a furyl fragment, the latter bridging in a sigma,pi-vinyl fashion. The molecular structures of 3, 5 and 6 have been determined by X-ray diffraction studies.
One of the oldest and most widely used commercial enzyme inhibitors is aspirin, Formula: C8H12BrN, which selectively inhibits one of the enzymes involved in the synthesis of molecules that trigger inflammation. you can also check out more blogs about 19977-51-6
Reference：
Piperidine – Wikipedia,
Piperidine | C5H15034N – PubChem